RR intervals were longer around self-terminating apnoeas/hypopnoeas (RR 914☒9 ms) the differences were not significant compared with undisturbed sleep. The LF and TF power increase was greater around arousal-inducing (LF 260±45 ms 2, TF 390☖5 ms 2) compared with self-terminating (LF 161☓1 ms 2, TF 249±40 ms 2) apnoeas/hypopnoeas the LF and LFn increases were significant in both groups compared with undisturbed sleep and HF power differences were nonsignificant. RR duration did not change around apnoeas/hypopnoeas (RR 904☒8 ms). LF increase was proportionally higher than the HF increase (normalised LF (LFn) 67☑ units, normalised HF (HFn) 33☑ units) compared with undisturbed sleep (LFn 52☒ units, HFn 48☒ units). The increase in high frequency (HF) power was not significant. Low (LF) and total (TF) frequency power increased 2 min around the end of apnoeas/hypopnoeas (LF 229☓8 ms 2, TF 345±45 ms 2) compared with undisturbed sleep (LF 106☑8 ms 2, TF 203☒3 ms 2). Fourteen untreated OSAHS patients and seven healthy subjects underwent overnight polysomnography. In the present study, the hypothesis was that sympathovagal balance around apnoeas/hypopnoeas and nocturnal autonomic activity are altered in OSAHS patients.įrequency- and time-domain analyses of RR intervals were performed to monitor sympathovagal activity noninvasively. A recent study has shown that daytime heart rate variability is reduced in obstructive sleep apnoea/hypopnoea syndrome (OSAHS) patients.
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